Most scientists think that SARS-CoV-2 underwent a natural process and evolved into a virus that could infect humans. The widely accepted theory is that, against all the odds, a bat coronavirus managed to get inside a pangolin and then get inside the exact same blood cell as a pangolin coronavirus, and then these two viruses gave rise to a brand new kind of virus that could infect humans. This is classed as zoonotic infection – this is where a virus is supposed to jump species and start being able to infect humans by acquiring specific mutations.
Over the last 20 years, there have been loads of outbreaks where a virus has jumped species and become a threat to humans – apparently, it just keeps on happening! Flu from pigs and birds…. SARS, MERS, and Ebola from bats.i But some scientists have dismissed the zoonotic theory for the origin of SARS-CoV-2; they think the bat/pangolin theory is very unlikely, for a whole bunch of reasons. These scientists are molecular biologists and geneticists who have studied the design of the virus and come to the conclusion that SARS-CoV-2 was carefully crafted in a laboratory, and that it either escaped from a lab accidentally or was released on purpose.
According to Yuri Deigin (Lab-Made? SARS-CoV-2 Genealogy Through the Lens of Gain-of-Function Research):
“If you hear anyone claim ‘we know the virus didn’t come from a lab’, don’t buy it — it may well have. Labs around the globe have been creating synthetic viruses like CoV2 for years. And no, its genome would not necessarily contain hallmarks of human manipulation: modern genetic engineering tools permit cutting and pasting genomic fragments without leaving a trace. It can be done quickly, too: it took a Swiss team less than a month to create a synthetic clone of CoV2.”
Deigin originally assumed it was bound to be a natural virus, and set out to prove the ‘conspiracy theorists’ wrong, but the more he looked, the more he started thinking they might be right, and that SARS-CoV-2 really could be a man-made virus. He says it’s “an obvious chimera” that’s based on a bat-CoV in which part of the spike (the RBM) has been replaced with the RBM from a pangolin-CoV. He also notes the unusual addition of a furin cleavage site which “significantly expands the ‘repertoire’ of the virus in terms of whose cells it can penetrate.”
This furin cleavage site is said to make the virus even more dangerous because of the way it gets processed by the body, but nobody can explain how it got there. It’s not found in similar coronaviruses so scientists can’t explain how SARS-CoV-2 came to have one. Lots of other viruses have got one, though, and the NIH came up with a solution called the pre-fusion technique, then spent several years perfecting the technique in secret tests with Moderna. In fact, all but one of the alleged outbreaks that have taken place over the last 40 years have involved a virus with a furin cleavage site. This includes HIV, influenza, MERS, Zika and Ebola; it was just SARS-CoV that didn’t have one. Moderna was secretly working with the NIH to develop vaccines for some of these viruses and this involved using viruses AND furin for testing.ii Reseachers have previously experimented with inserting a furin cleavage site into SARS-1 (e.g. in 2006 and 2008).
One of the most important reasons to study the design of the rona is that most of the changes that have aroused suspicion involve the spike protein, and it’s the spike protein that’s in the vaccines. A key feature of the spike is the RBD (receptor binding domain) because this is the bit that determines how a virus is able to infect a particular species, and the RBD of the rona is very suspicious indeed. It just doesn’t seem natural.iii It’s supposed to have evolved from a bat virus, and a pangolin virus, so it should be able to infect bats and pangolins better than it can infect humans, but in fact the SARS-CoV-2 virus is far better at infecting humans than any other species!